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 • Scientific Committee
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• PS1 Physiology
• PS2 Pathophysiology
• PS3 Investigations
• PS4 Investigations (Cont.)
...........Symptomatology
• PS5 Extra-esophageal symptoms
...........ENT symptoms
• PS6 Pulmonary symptoms
• PS7 Histology
...........Medical Treatments

PS1 Physiology Monday, September 1, 2003
Presidents:J.E. RICHTER (USA)

M. HONGO (Japan)
8:30 a.m.
 Is duodeno-gastric reflux (DGR) a physiologic event with varying composition?
K.H. FUCHS (Wurzburg)

From the information given by esophageal pH, impedance monitoring and Bilitec probe, what is the rate of non-acidic reflux in normal subjects?
D. SIFRIM (Leuven)

What is the proportion of duodenal content in non-acidic reflux?
J. TACK (Leuven)


8:45 a.m.
9:00 a.m.


R.K. MITTAL (USA)

What are the means of secretion of chloride and bicarbonate in the duodenum?
S.K. KONTUREK (Krakow)
What is the role of PGs in the mediation of the process of secretion?
S.K. KONTUREK (Krakow)
What is the role of H pylori in the PGE2 stimulated H CO3 secretion?
M. BLASER (New York)


9:15 a.m.
9:30 a.m.


C. SCARPIGNATO (Italy)

What are the compared mechanisms of cellular defense in the esophagus and at the laryngeal level?
N. JOHNSTON (Winston Salem)

What criteria can allow for the diagnosis of pathological duodeno-gastro-esophageal reflux (DGER)?
J. TACK (Leuven)

Can preservation of the clearance mechanisms prevent the development of esophagitis in patients with DGER?
K. GEBOES (Leuven)

Can the role of the antro-pyloric motor activity in preventing DGR be assessed ?
A. SMYTHE (Sheffield)

9:50 a.m.
Topic Forum
 Do acid and DGER act synergistically in the development of complications of GERD?
P. BIANCANI (Providence) - N. BUTTAR (Rochester - MN)
10:05 a.m.



10:10 a.m.
 PAUSE